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TRM Labs

TRM Labs

A regulatory technology company offering a transaction monitoring solution to prevent fraud on digital assets companies. It utilizes behavioral analytics to detect suspicious anomalies.

TRM Labs is a regulatory technology company offering a transaction monitoring solution to prevent fraud on digital assets companies that is headquartered in San Francisco, California and was founded in 2017 by Esteban Castaño and Rahul Rana. The company is utilizing behavioral analytics to detect suspicious anomalies to create a platform for institutions to fight back against money laundering, terrorism financing, fraud, and breaches of compliance.

Funding
Seed

On January 31, 2019 TRM Labs completed their seed funding round with $1.7 million in funding from Blockchain Capital (lead investor), The MBA Fund, Green D Ventures, and Alumni Ventures Group.

Timeline

December 7, 2021
TRM Labs raises a $60,000,000 series B round from American Express Ventures, Citi Ventures, DRW, Jump Capital, Tiger Global Management and Visa Inc..
June 17, 2021
Blockchain analytics firm TRM Labs raises $14 million Series A funding round led by Bessemer Venture Partners.
June 17, 2021
TRM Labs raises a $14,000,000 series A round from Blockchain Capital, Initialized Capital, Jump Capital, Operator Partners, PayPal Ventures, SGH Capital, Salesforce Ventures, Tapas Capital, The MBA Fund and Y Combinator.
November 19, 2019
TRM Labs raises a $4,200,000 seed round from Blockchain Capital, Initialized Capital, PayPal Ventures, SGH Capital and Y Combinator.
January 2019
TRM Labs raises a $1,700,000 seed round from Blockchain Capital, Green D Ventures, Tapas Capital and The MBA Fund.

Funding rounds

Patents

Further Resources

Title
Author
Link
Type
Date

News

Title
Author
Date
Publisher
Description
Angus Liu
November 6, 2020
FierceBiotech
Elderly survivors of COVID-19 and influenza are prone to long-term lung problems. A new study from the Mayo Clinic offers one possible explanation for why older people are more susceptible to lung inflammation and fibrosis following viral infection--and tissue-resident memory CD8+ T cells might be to blame.

References

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